Canadian Forest Service Publications

Sudden Oak Death (Phytophthora ramorum): Research Update, Risk Analysis and Mitigation Approach for Canada. (Abstract) 2011. Shamoun, S.F.; Rioux, D. Pages 24-25 in "Responding to Invasive Species" 2011 Research Forum, October 18-19, 2011, Richmond, BC. Invasive Plant Council of British Columbia, Williams Lake, BC.

Year: 2011

Issued by: Pacific Forestry Centre

Catalog ID: 32871

Language: English

Availability: PDF (download)

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Phytophthora ramorum (Pr) is an exotic pathogen causing a devastating disease known as sudden oak death (SOD) (= ramorum bleeding canker) of oak and tanoak trees in native forests of the western US. It also causes ramorum leaf blight or ramorum shoot dieback of woody ornamentals such as rhododendron and camellia in forests, retail or wholesale nurseries, and garden environments. This pathogen can infect more than 120 hosts, several of which being present in Canadian forested and urban areas. In Canada, several nurseries in BC have reported Pr-infected plants, and eradication measures have been taken with success to prevent the spread of the pathogen. Canadian Food Inspection Agency has completed its 2010 national survey for Pr. Of the 133 nurseries sampled throughout Canada, Pr has been detected at five retail nurseries and two propagation nurseries in urban areas of southwest BC. Research activities at the Canadian Forest Service have been mainly carried out to better understand the biology, population genetics, and mitigation measures to help assess the risk associated with Pr in Canada. Our presentation will summarize research results on: 1) development of PCR-DNA molecular markers to identify the three (2 North American and 1 European) Pr lineages; 2) efficacy of commercial biological control products and fungicides against Pr; 3) assessment of the aggressiveness among the three lineages of Pr; 4) evaluation of susceptibility to Pr of nursery plants, forest vegetation and tree species common to eastern and western Canada; and 5) research of putative resistance mechanisms in trees to Pr.